Several studies have demonstrated that HMGA1 proteins play a causal role in neoplasticcelltransformation.
2
Treatment of these cells with Mith also decreased Mcl-1 expression and neoplasticcelltransformation, and this was accompanied by induction of several markers of apoptosis.
Ús de dysplasia en anglès
1
Conclusion: High-grade dysplasia diagnosis was inconsistent in more than half of patients.
2
Results: There were no differences in dysplasia recurrence between the 2 groups.
3
We analyzed a family with an autosomal recessive form of bone dysplasia.
4
These results support a dichotomy in the dysplasia-CIS sequence in the pancreas.
5
In some cases dysplasia reappeared after a long-time interval of apparent regression.
6
The microscopic findings suggest the lesion is possibly of a collagenous dysplasia.
7
Microscopic examination of the FA patient's OPMD confirmed the presence of dysplasia.
8
To date, there has been no curative therapy for this skeletal dysplasia.
9
Objective: The cortex of patients with cortical dysplasia contains several abnormal cell types.
10
Results: Nonlethal skeletal dysplasia was suspected and then diagnosed after 17 gestational weeks.
11
The management of patients with high-grade dysplasia without evidence of carcinoma remains controversial.
12
We used three-dimensional tissue culture to build an organotypic model of bronchial dysplasia.
13
Eradication of dysplasia and Barrett's esophagus was the main outcome measure.
14
No cases with mild dysplasia were classified as severe dysplasia and vice versa.
15
Histopathologic analysis by an experienced bone pathologist confirmed fibrous dysplasia in all patients.
16
Histologic grading of dysplasia using established criteria is a powerful prognosticator in BE.